Inherency and Food Addiction, Continued

The previous post mentioned some of the authorities who have a hard time justifying a scientific basis for the intrinsic addictiveness of foodstuffs. But always, there is just enough evidence to make the possibility tantalizingly attractive to many parties, for various reasons.

The title “The Biology Behind ‘Food Addiction‘” puts the loaded term in quotation marks, and discusses the activation of reward systems:

[T]he activity of the endogenous, natural, opioid system is influenced by ingestion of palatable diets, and […] changes in the activity of the system in turn affect behavior, feeding and, perhaps, diet preference.

Similarly, both dopamine release and dopamine receptor levels are affected by palatable diets. From this it is apparent that dysfunction of these systems has the potential to contribute to overeating and the pathophysiology of obesity.

The authors explain that brain reward systems evolve to reinforce natural behaviors that are advantageous to the individual. Addictive substances connect up with the same reward systems and subvert them, making their existence a liability rather than an asset. Still, this does not constitute convincing proof that foods or food components should be considered addictive in the same sense as, for instance, cocaine.

The same organization published in 2016 a multi-faceted report on their investigations into “clinical, basic, and epidemiological research exploring the neurobiological interface between food intake, reward and stress.” They found slender evidence to support the idea that food or isolated ingredients can cause addiction in the same sense as addictive drugs.

Sugar probably presents the strongest case for thinking of a food as addictive, but the notion of sugar addiction left this team unmoved for a number of reasons:

[W]e find little evidence to support “sugar addiction.” Indeed, in animal studies we found divergence regarding brain pathways involved in reward behavior for a sweet reward from those that specifically modulate addiction-related behaviors.

The authors went on to speak of the importance of interactions between the brain’s reward pathways and the regulatory circuitry in the body that pertains to feeding. They mention the poor understanding of the neural substrate that makes this all happen, or not happen, as the case may be. It is known that the hormones leptin and ghrelin signal for other things to occur, but lining up their actions with the origin and persistence of eating disorders was nowhere near complete enough to be positive about anything.

In summation,

There is a reasonable expectation that this interaction is key to the addictive properties of specific food components and the emergence of disordered eating. But it is poorly understood how food components affect the reward circuitry, and to what extent hormones provide the link between ingestion of food and reward, and we have a poor understanding of the addictive properties of individual food components.

Your responses and feedback are welcome!

Source: “The Biology Behind ‘Food Addiction'”, Cordis.europa.eu, undated
Source: “Final Report Summary — NEUROFAST (The Integrated Neurobiology of Food Intake, Addiction and Stress.),” Cordis.europa.eu, January 2016
Image by Robin Stickel on Unsplash 

Inherency and Food Addiction

Debate continues about whether some foods can be addictive in the same sense as, for instance, heroin. Is the addictiveness inherent in the food, or in the person? Or does it have to be both, two variables with the potential to cause damage, and when they meet up, the person is doomed to a lifetime of struggle?

A 2020 paper by Dr. Pretlow and three co-authors says,

There is increasing interest in whether overeating/obesity stems from an addictive process, although this notion is controversial… Food addiction (FA) connotes a substance dependence on ingredients in food, e.g., sugar, and is comparable to drug and alcohol dependence. The FA construct involves addictive eating of certain foods, which are craved, sought out, and eaten in excess.

At the same time, there is “a dearth of evidence supporting substance dependence on food ingredients.” For starters, all foods do not have the same power over people, which opens the way for facetious questions like, “Why there are so many chocolate addicts and so few broccoli addicts?” To complicate matters even further, legit substance dependencies like drug and alcohol addiction also have behavioral components. Even years ago, an important point was being made:

Labeling a food or nutrient as “addictive” implies that it contains ingredients and/or possesses an inherent property with the capacity to make susceptible individuals addicted to it, as is the case for chemical substances of abuse.

In practical terms, inherency means that no matter how expensive a substance may be, people will buy it. They will stick a hypodermic syringe between their toes to get it into their bloodstream, and risk imprisonment and disgrace. In food terms, they would eat it whether they were hungry or not, and even if the stuff tasted awful. If it meant going to jail, they would do it anyway. In all honesty, it would be difficult to name a food with those properties.

A 2014 paper encouraged professionals in the field to begin thinking in terms of “Eating Addiction.” Even though in practical terms, such foods as corn, wheat, coffee, milk, eggs, and potatoes do a very good job of making a case for addictiveness, there seems to have been difficulty in identifying any chemical or “substance-based” causative agent.

Officially, this was the state of affairs at the time:

The currently available evidence for a substance-based food addiction is poor… Humans who overeat usually do not restrict their diets to specific nutrients; instead the availability of a wider range of palatable foods appears to render prone subjects vulnerable to overeating.

The new DSM-5 (APA, 2013) currently does not allow the classification of an “Overeating Disorder” or an “Addictive Eating Disorder” within the diagnostic category Substance-Related and Addictive Disorders; indeed, the current knowledge of addictive eating behaviors does not warrant such a diagnosis.

[T]here is currently insufficient scientific evidence to label any common food, ingredient, micronutrient, standard food additive or combination of ingredients as addictive.

Your responses and feedback are welcome!

Source: “Treatment of the sensory and motor components of urges to eat (eating addiction?): a mobile-health pilot study for obesity in young people,” Springer.com, 01/14/20
Source: “’Eating addiction’, rather than ‘food addiction’, better captures addictive-like eating behavior,” ScienceDirect.com, November 2014
Image by whologwhyCC BY 2.0

Some Wrinkles in the Food Addiction Concept

Many aspects of the food addiction (FA) concept can be discussed extensively, including whether we are talking about a substance addiction or a behavioral addiction, or both, or neither; and even whether there are more than two main camps.

Discussing FA is tricky because a good argument can be made that it is a behavioral addiction to one or more of an assortment of displacement behaviors including biting, chewing, licking, sucking, crunching, swallowing, etc. Yet at the same time, it is undeniable that what these people eat is food. Sure, in a few exotic cases people cannot stop eating styrofoam or mattress stuffing. But for the vast majority, what they are eating is food (or at least it is marketed as such).

One belief is that what is called food addiction is actually a subset of drug addiction. While some edible substances apparently have a drug-like effect on the brain, this is by no means a blanket truth. The conditions characterized as sex addiction and love addiction also produce endogenous chemicals like oxytocin, but relating this to someone who scarfs down a pound of potato chips at one sitting is a stretch.

In “The View from Rat Park,” Bruce Alexander explained how rats who had morphine available consumed a lot more of it when they were isolated:

This fact definitely undermined the supposed proof that certain drugs irresistibly cause addiction. [T]he drug only becomes irresistible when the opportunity for normal social existence is destroyed.

Under such conditions, both rats and people consume too much of whatever drug is made easily accessible to them. [I]t is not too early to be sure that the old theory that addiction is a problem caused by addictive drugs is far too simple…

Moreover, it has become absolutely clear that drug and alcohol addiction is only a corner of a much larger addiction problem!

A basic difficulty in this type of study is that lab subjects and lab conditions are very different from real life. Of course, lab conditions must be strictly controlled, and much discipline must be observed. Some argue that there is no way to know what rats are feeling, which is a good point. We can only presume from the way they act. Rat Park showed that the closer the conditions are to real life — i.e., replete with variables — the more difficult it becomes to tease out meaningful differences in response to any single stimulus.

When trying to measure or observe something, a slew of variables will definitely complicate matters. Rat Park showed what happens when variables are introduced. A creature with the potential to become a junkie is presented with the opportunity to take another path. There are toys, frisky playmates, and intriguing corners to hide in. When the environment contains things other than a morphine dispenser, the world is a different place, and you get a different kind of rat.

Some researchers have invested a great deal of credence in the idea that there is an orderly and inevitable progression from trying a substance for the first time to becoming a bona fide addict. As H. Ziauddeen and P. C. Fletcher put it,

Seminal models of drug dependence have characterized a set of core processes involved in the transition from drug taking to drug dependence. There is little consistent data across these various studies and the findings thus far do not support an addiction model or indeed any one model of altered brain function in obesity.

In “Is food addiction a valid and useful concept?” the authors concluded that evidence for its existence in humans is actually rather scarce, and it is “clear that an addiction model has a limited, if any, place in understanding obesity.”

Your responses and feedback are welcome!

Source: “Addiction: The View from Rat Park,” BruceKAlexander.com,
Source: “Is food addiction a valid and useful concept?,” NIH.gov, January 2013
Image by Matt/CC BY-SA 2.0

Different Schools of Thought

Late in 2014, a lengthy contribution was added to the conversation about terminology. ” ‘Eating addiction’, rather than ‘food addiction,’, better captures addictive-like eating behavior” is the title of a report published by Neuroscience & Biobehavioral Reviews. The authors (13 in number) note that some people believe their relationship with problem foods constitutes an addiction, and if they seek help, they look to the addiction sector of therapeutic possibilities.

They also mention that food addiction means different things to different people, who are talking about either substance addiction or behavioral addiction, or both, and this ambiguity causes confusion. They prefer “eating addiction,” and proceed to explain why.

Observations

First, they say there is no evidence that it is a substance use disorder. Sure, there have been indications that the brain equally welcomes a rush from cocaine and an ecstatic experience from the chocolate sauce. But by and large, it has been difficult to pinpoint addictive substances in the food itself, although, of course, some results have been quite clear. But no one has tried to make the case that drug-like substances are in every type of food that obese people eat.

While they scoff at the idea that the substance use disorder category of the DSM-5 will ever recognize “Glucose/Sucrose/Fructose Use Disorder” as a diagnosis, they also concede that both rodent and human data are consistent with the existence of addictive eating behavior. Landing somewhere in the middle, they agree with contemporary experts who say it is “premature to conclude validity of the food addiction phenotype in humans from the current behavioral and neurobiological evidence gained in rodent models.”

Let’s roll with it

But for the time being, and because it works adequately for educational purposes, they will go with “eating addiction” because it highlights the behavioral component. The point of this paper is to discuss, from the behavioral, clinical, and neurobiological angles, how addiction and overeating are both the same and different.

And again, the terminology becomes the subject. Where is the line between occasional overeating and binge eating? In what ways does an eating addiction resemble binge eating (or not)? How do we make sense of addictive disorders that are not even substance-related?

Like so many other problems, overeating is multifactorial. The pros have to look at the severity, the degree of compulsiveness, and the “clinically significant level of personal impairment.” These authors regret that Binge Eating Disorder has become conflated with food addiction, for this reason:

The impaired control over eating behavior in “eating addiction” does not necessarily require that the affected individual experiences a sense of lack of control over eating during a single episode of overeating.

Your responses and feedback are welcome!

Source: “‘Eating addiction’, rather than ‘food addiction’, better captures addictive-like eating behavior,” ScienceDirect.com, November 2014
Images by Jesse Case and CC Runs via Twitter

Moderation Is or Is Not the Answer

Dr. Pretlow says,

A central barrier to the success of treatment for obesity that is distinct from drug addiction, is the fact that food consumption is essential for survival; thus, abstinence is not a feasible or appropriate treatment goal.

But what about moderation? In discussions of addiction and recovery, one of the first principles is that moderation fits in there somewhere. However, some believe it works, and others say it is impossible.

People in the eating disorder community, or the diabetes community, are all about eating normally. That is their goal. But an alcoholic can’t pursue the goal of “normal” social drinking, like two cocktails a week or whatever, because addiction does not work that way. “Harm reduction” or “moderate drinking” does not work for addicts.

Billi Gordon, Ph.D., who was a Research Associate at UCLA’s Center for the Neurobiology of Stress and Resilience, was never shy about sharing his own experiences, including his weight which at one point reached almost 1,000 pounds. In a Psychology Today piece called “Moderation — Strategy or Fantasy?” he wrote,

I am a food addict — and barbecue is my favorite fix… My ultimate comfort food.

In other words, his #1 problem food. This was because of childhood memories of social approval and attention at family gatherings, and particularly of time shared with his father. As he aged, this need for connection did not fade, but became stronger:

[T]he dependence on the symbolic interaction with the comfort food grows. So finding replacements that completely satisfy the needs your symbolic interaction with comfort foods only partially satisfied, is how you achieve moderation…

Sure, on one or more holidays he tried saying no to barbecue, and the results were severe depression and bingeing. Apparently, such an iconic comfort food cannot be replaced, as nothing else is capable of satisfying the symbolic interaction need.

In addition, Dr. Gordon had looked up behavioral studies on goal achievement, which showed that the more specific a goal is, the more achievable it is. He concluded that moderation is “very unspecific and determined by what one’s brain considers as normal,” a goal too nebulous to be effectively pursued. He wrote, “I need a strategy, not a fantasy, and moderation is a fantasy — at best.”

Dr. Gordon wrote extensively on the similarity between binge eating and compulsive eating, and drug addiction. He suggested an “underlying neurobiological process similar to addiction.” Dopamine, of course, is involved, activating the brain’s reward areas, and repeated indulgence in stress-relieving problem foods alters the dopaminergic pathways. He wrote:

[D]rug and food stimuli cause the same type of conditioned gene expression and neuronal plasticity in the mesolimbic-cortical pathway (reward circuitry) and regions associated with learning and memory, e.g., the ventral striatum, where habit formation occurs. Dopamine and endogenous opioids are implicated in adaptations to reward circuitry in compulsive overeating, as well as drug usage.

Your responses and feedback are welcome!

Source: “Moderation – Strategy or Fantasy?,” PsychologyToday.com, 07/06/16
Source: “Christmas Cookie Blue,” PsychologyToday, 12/06/13
Image by Kirt Edblom/CC BY-SA 2.0

Obesity and Addiction Debate Points

There are first principles that anchor the various concepts about addiction as it relates to obesity. Often there is no exact agreement on the principle itself; only on the importance of taking the matter into consideration.

One principle is inevitability, the conviction that if something in the environment is capable of causing addiction, people will get hooked. And sure, some do. But some don’t. This is one of the variables that can knock holes in theories. A closely related concept is that some foods are just inherently junkie fodder. In other words, an addictive food will be the downfall of anyone who is exposed to it. This view is held by some and contested by others. On both sides, the evidence is out there. Some people, confronted by the most delicious treats, are unmoved.

In “Addiction: The View from Rat Park,” Bruce K. Alexander threw shade on the Skinner box research “which once appeared to show that all rats and people who use addictive drugs become addicted.” Then along comes a doubter who asks why everybody isn’t addicted. Someone else says, if we look closely enough, they probably are, to something. A 2013 paper on the validity of the food addiction concept said,

The hyperpalatable foods that are thought to be addictive are widely available and widely consumed. To consider that they may become addictive in some individuals will require the characterization of a specific feature (or several features) of these foods that acts in concert with certain individual vulnerabilities.

Dr . Vera Tarman has pointed out that while anybody can become an addict, surprisingly, not everyone does — either to a substance that many are susceptible to, or under circumstances where others buckle. These are two of the anomalies that mess up attempts to achieve consensus. She has also posed many questions, like:

How about vegetables, fruits, meats and fish? For most people, even end-stage food addicts, these foods are not addictive.

She states that humans are programmed to want food when hungry, along with a complementary instruction to be satisfied when we have extracted the stored energy from the food. But that only works in relation to “the foods our body was metabolically designed to eat and enjoy with satisfaction.” When the border is crossed into the area of weird, manipulated food, however, that is another story. We live in an environment that creates food addicts on purpose.

In the words of Billi Gordon, Ph.D.,

You can never get enough of something that’s almost satisfying.

If this subject is to be debated thoroughly, there are a lot of side roads. One of the ironies is that, according to people who devote their lives to figuring out the best way to eat, there is an available way to fill the emptiness. They will affirm that something like a bowl of brown rice and beet greens can make a person “feel fed” in a way that cannot be described, but once felt, is unmistakable. The eating part itself may not provide quite the same thrill as a bag of cheese puffs, but the body as a whole really does receive the message: “Hey! I got some nutrients! I can quit eating for a while!”

Your responses and feedback are welcome!

Source: “Addiction: The View from Rat Park,” BruceKAlexander.com, 2010
Source: “Is food addiction a valid and useful concept?,” NIH.gov, January 2013
Source: “Guest Post: Food Abstinence for Food Addicts: Deprivation or a New Freedom?,” DrSharma.ca, February 2015
Source: “Moderation — Strategy or Fantasy?,” PsychologyToday.com, 07/06/16
Image by emanoellers/CC BY 2.0

Obesity and Addiction Debates

One of the first things that people need to do is figure out if they are even talking about the same thing. This is especially true if they are using the same word while maintaining different mental pictures of what it means. One choice would be to bring in a different word, but then the people who are talking about that word already might take offense because they mean something different by it. Hashing out the differences is important, and can be exhausting.

It appears to Dr. Pretlow, as well as to leaders in addiction science, that addiction and obesity both stem from one source. They both “reflect the consequences of ingestive behavior gone awry.” This is apparent from the core similarities shared by the two conditions:

First, in terms of clinical diagnostic features, both addiction and obesity result from repetitive foraging and ingestion behaviors that intensify and persist despite negative and (at times) devastating health and other life consequences.

Second, only a subset of individuals who are exposed to substances with addictive potential develop addictive behaviors, just as not all people who are exposed to foods and diet patterns that pose difficulties with weight control become obese.

The first item includes the words “behaviors that intensify and persist.” Classically, intensification and persistence are hallmarks of addiction. So it feels natural to include people who can’t or won’t stop eating, no matter what. But then, other questions arise. What exactly are they addicted to? A substance, a certain ingredient in certain foods? Or to the fundamental physical satisfactions, and the ceremonies, of eating itself?

One hallmark of addiction is that it tends to continue, and get worse over time. To proponents of either substance addiction or behavioral addiction, this is a primary tenet, part of the definition.

But then along came studies that pointed out an unexpected trait in at least two groups of people, returning Vietnam vets who had been “self-medicating” while stationed overseas, and many stateside patients who had been legitimately medicated after surgeries. A large number of individuals in each category were found to be able to give up opioids with the ease of shaking the dust off a hat. Until somebody figures out that kind of mystery, how can dialogue about substance versus behavior even be initiated?

Subset of individuals

The second item covers part of that same ground. Not only are some people able to easily abandon what appeared to have been addictions; some people never catch habits in the first place. As Dr. Pretlow points out, not all who are exposed to potentially addictive substances will develop addictive behaviors — “just as not all people who are exposed to foods and diet patterns that pose difficulties with weight control become obese.” The surprising Rat Park studies revealed that, given alternative activities and some kind of basically satisfying lifestyle, even lab rodents might turn down the opportunity to become addicted to hard drugs.

The existence of so many anomalies is the source of unease in both the Food Addiction and the Behavioral Addiction schools of thought. It becomes tempting to ask, only half-facetiously, “Maybe the people are addicted to neither food as a substance, nor eating as a behavior, but to obesity itself?” Because on some days, that seems to make as much sense as anything else.

Your responses and feedback are welcome!

Image by Jason Wilson/CC BY 2.0

Withdrawal’s Big Problems

The previous post talked about withdrawal, which sometimes, even in the case of legitimately prescribed pharmaceuticals, can be a full-scale life-threatening experience requiring medical support; or perhaps only put a patient through a period of malaise and general discomfort. In any case, subjective experience is a difficult thing to argue. Still, as long as there are people who believe that quitting problem foods is comparable in misery to quitting a hard drug, it is certainly a problem to be dealt with.

Some formerly obese people have reported cravings for a while, until the body readjusts to the new regime. Childhood Obesity News has mentioned Dwight Riskey, who is a cravings expert. He worked with a team at Monell Chemical Senses Center which found that “people could beat their salt habits simply by refraining from salty foods long enough for their taste buds to return to a normal level of sensitivity.” Sounds easy!

One school of thought holds that along with letting withdrawal happen, it is important to take active detox measures at the same time, and there are versions of this plan for every substance including food. Philip Werdell wrote extensively on the subject. When asked what happens, once a food addict begins treatment, he said:

When overeaters are separated from their primary binge foods, the first change is that they stop having physical cravings or, at minimum, the cravings are lessened to the point where they are no longer overpowering.

Even more important for long-term success, the mind of the detoxified food addict begins to change in remarkable ways. Where once they believed their own rationalizations (read: lies) about food, detoxification helps the food addict begin to see his or her past thinking as distorted.

Beat, a British organization for people with eating disorders, provides space for peer support and online development, and also works to inform the government about what kinds of backing and research are needed if the nation’s obese people are actually to be helped. Spokesperson Frankie Mullin described addicted people’s problem as “trying to fill a bottomless void and no amount of food will satisfy their cravings.”

Withdrawal from alcohol leads to a state of sobriety, which some people find very unpleasant indeed, and also compare to an endless chasm of emptiness. When writer Benjamin Davis got sober, he felt constant boredom, inability to sleep, and social anxiety, concluding that “if I didn’t find ways to solve those problems, I’d be back to drinking again.” Worse, he felt he had been misled, tricked into thinking that sobriety would cure all his problems, only to find that “actual solutions take time, patience, and continuous effort.” He started reading for several hours per night, dropped people who did not add value to his life, and conveyed the message to supportive friends:

People need more than support; they need solutions, alternatives, and creative thinking. Otherwise, best case scenario, we just get addicted to something else…

Your responses and feedback are welcome!

Source: “The Extraordinary Science of Addictive Junk Food,” NYTimes.com, 02/20/13
Source: “How emotional eating keeps you stuck,” OhThatsTasty.com, undated
Source: “Food addiction: know the facts,” Food.UK.MSN.com, 01/05/2013
Source: “The Art of Staying Sober,” medium.com, 07/25/21
Image by gunman47/CC BY-ND 2.0

Is Withdrawal a Thing?

There is a term, “definition monopoly,” meaning that somebody has the sole right to say what a word or expression means. Apparently, food addiction/eating addiction are areas where consensus is an elusive goal.

Some professionals believe that the term “withdrawal “should not be used in reference to food at all, because it is not a physically addicting drug, and because the problem is not the food per se, but the behavior of eating it inappropriately. It is also said that in psychological addictions, there is no such condition as withdrawal or tolerance buildup.

As we have seen, others, like Zoe Harcombe, find the concept useful. In listing the four stages of food addiction she writes,

3) We feel bad when we don’t have the food – we literally get withdrawal symptoms in the absence of our fix.

And from the other end of the spectrum, there is evidence that some populations habituated to opioids, including medical patients and Vietnam veterans, dropped their habits with very low or no consequences, once the time of acute need had passed.

The misnomer problem

Withdrawal seems to have a fairly nebulous meaning. Getting off some drugs (including prescriptions) can be a life-threatening ordeal, requiring medical supervision. Meanwhile, jokes are made about having “withdrawals” from one’s favorite peanut butter and banana sandwiches. At any rate, the topic seems worthy of discussion, because opinions differ.

To get to a diagnosis of addiction, some say, the presence or threat of an extremely painful experience must be a necessary condition. This is the trauma of physical withdrawal, as depicted in movies that are meant to scare viewers straight.

In online forums where regular people discuss their experiences, it is possible to find extreme statements, like a description equating the first few days of a diet to withdrawal from heroin. And who knows? Maybe it is like that. Maybe it is presumptuous to say anything about other people’s pain.

Letting go

As we have mentioned, the group FARE (Food Addiction Research Education) certainly acknowledges withdrawal symptoms as a very real and serious factor that prevents people from trying to end their dangerous dependency on the wrong kinds of food and too much of it, which in turn pulls them into serious problems like obesity, diabetes, and heart disease. FARE says,

Withdrawal symptoms can include severe anxiety, headaches, sadness, anger, sweating, shaking, disorientation and depression. They can last anywhere from days to weeks or even months after quitting.

Despite a strong desire to stop, the complexity of physical withdrawal symptoms and the accompanying emotions can lead an individual back to using mood-altering foods, which only perpetuate the addiction cycle.

Your responses and feedback are welcome!

Source: “What exactly is withdrawal?, FoodAddictionResearch.org, undated
Image by Nick Fisher/CC BY-SA 2.0

Relapse — Word of Dread

With a substance addiction, like nicotine, total abstention is a possibility, and a total quit gives a person much better odds of not relapsing. A cliché is that someone who has not smoked in years can be rehooked with astonishing rapidity. Even when people try hard, the relapse rate is discouraging. With certain substances, like cocaine and alcohol, moderation cannot be the answer in the overwhelmingly vast majority of cases. Quit means quit.

With food, the inability to totally abstain is the hardest thing. Someone compared it to divorce. With alcohol, heroin and many other substances, it’s like a divorce without kids. You can just say “We’re done” and never have to see the person again. But if there are children, you have to deal with shared custody and child support and then the grandkids… You’re tied to your ex forever. And common decency does not allow cutting them out of your lives like a bad habit.

With disordered eating, what forbids a person to walk away is basic survival. Sometimes, beating a food habit requires a great deal of ingenuity, or it might wreck a life in other ways. To consume food is our fate, but it does not have to be our doom.

Moderation is not an option

There is plenty of discussion about food addiction versus eating addiction, whether some foods are inherently addictive, and many other variables. Still, no matter what philosophy a striving person chooses or what program they choose, relapse is always an open door that extends the invitation to “walk right in, set right down.” Dr. Pretlow has written,

Despite often repeated attempts to reduce or quit using addictive substances, relapse is common in the addiction recovery process, just as those with obesity who attempt to regulate their food intake through dieting frequently relapse and return to their elevated body weight.

Just last month, writer Addy Baird looked back and described her young life as “largely defined by my obsessive food and body rituals.” Baird notes that studies hope to hone the definition of what eating disorder recovery actually looks like, gauged by “a combination of physical, behavioral, and psychological indicators.” She then goes on to say,

Notably, one study found that a group considered “fully recovered” had similar results to a control group, but elevated rates of anxiety disorders.

She quotes therapist Carolyn Costin, who defined recovery as accepting one’s body and not having a “self-destructive or unnatural relationship with food or exercise.” In Costin’s words,

When recovered, you will not compromise your health or betray your soul to look a certain way, wear a certain size, or reach a certain number on a scale.

Baird also shared with and learned from a sobriety community and especially from Michelle Callahan, a substance abuse treatment therapist who rejects the limited duality of complete recovery/abject relapse. She prefers the gentler word “lapse” and speaks of a flexible type of ongoing, ever-present recovery that may include a lapse now and then.

Callahan wants people to understand that not all steps point forward, and to never get overwhelmed, or think that all their hard work was for nothing, and especially never to feel shame. Here is the crux of the matter:

They can also feel like they are a failure, are bad, or weak, which are likely the core beliefs that pushed them into addiction or engaging in their problematic behaviors in the first place.

Instead, it can be useful to regard a lapse as a pop-up research lab that teaches the person what not to do next time. Addy Baird wrote,

As my healthy self got stronger and my relationship to my food and exercise began to transform, it became clear that giving myself more space to learn (and sometimes fail) was vital.

Your responses and feedback are welcome!

Source: “Giving Up On A Perfect Recovery Actually Helped Me Heal From My Eating Disorder,” BuzzFeedNews.com 03/25/22
Image by dynamosquito/CC BY-SA 2.0

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Profiles: Kids Struggling with Weight

Profiles: Kids Struggling with Obesity top bottom

The Book

OVERWEIGHT: What Kids Say explores the obesity problem from the often-overlooked perspective of children struggling with being overweight.

About Dr. Robert A. Pretlow

Dr. Robert A. Pretlow is a pediatrician and childhood obesity specialist. He has been researching and spreading awareness on the childhood obesity epidemic in the US for more than a decade.
You can contact Dr. Pretlow at:

Presentations

Dr. Pretlow’s invited presentation at the American Society of Animal Science 2020 Conference
What’s Causing Obesity in Companion Animals and What Can We Do About It

Dr. Pretlow’s invited presentation at the World Obesity Federation 2019 Conference:
Food/Eating Addiction and the Displacement Mechanism

Dr. Pretlow’s Multi-Center Clinical Trial Kick-off Speech 2018:
Obesity: Tackling the Root Cause

Dr. Pretlow’s 2017 Workshop on
Treatment of Obesity Using the Addiction Model

Dr. Pretlow’s invited presentation for
TEC and UNC 2016

Dr. Pretlow’s invited presentation at the 2015 Obesity Summit in London, UK.

Dr. Pretlow’s invited keynote at the 2014 European Childhood Obesity Group Congress in Salzburg, Austria.

Dr. Pretlow’s presentation at the 2013 European Congress on Obesity in Liverpool, UK.

Dr. Pretlow’s presentation at the 2011 International Conference on Childhood Obesity in Lisbon, Portugal.

Dr. Pretlow’s presentation at the 2010 Uniting Against Childhood Obesity Conference in Houston, TX.

Food & Health Resources